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Am J Respir Crit Care Med , 2006, 174(7): 810-816

Some studies suggest that the resistance mechanisms of c-MET inhibitors usually fall into one of two categories: (i) target resistance mediated by secondary MET kinase domain mutations and/or allele amplification of METex14

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Lysine residues in histones undergo post-translational modifications such as acetylation and methylation, regulating gene expression