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Under neuropathological conditions, mitochondrial defects often lead to electron transfer processes and reduced NADPH oxidase activity, resulting in increased reactive oxygen species production, which weakens the redox buffering capacity of the cell and may damage key enzymes involved in energy metabolism (Tang, 2020)

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Figure 3 Additionally, MB demonstrated a moderate citotoxicity profile against hepatocarcinoma cell line HepG2, yielding an EC 50 of 2.68 1.81 M ( Supplementary Material Figure 1 )