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Thus, by inducing GPx1 expression p53 increases the cellular anti-oxidative capacity when DNA damage is detected

Blood samples were collected 1 h after each second injection (at 12, 24, and 48 h), and serum uric acid concentrations were analyzed to assess treatment efficacy and monitor hyperuricemia progression

The headline mechanisms: Angiogenesis via VEGFR2/Akt/eNOS: This is clinically significant because tendons and ligaments are notoriously hypovascular

However, as an emerging discipline, the preparation and characterization, targeted delivery, and controlled release of nanomaterials remain a challenge, and the issues of biosafety, efficacy complexity, cost, and regulation also need to be optimized