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In many disease models, such as those representing joints, metabolic organs, or immune niches, replicating in vivo -like crosstalk requires the co-culture of distinct patient-derived cells, including epithelial, stromal, immune, and vascular components

Acknowledgements This work was supported by the National Natural Science Foundation of China (82273092) to LFF and Natural Science Foundation in Zhejiang province (LD22H160003) to XW

Epub 2021 Nov 15

These signaling pathways are initiated by ischemia-induced hypoxia and oxidative stress and are propagated through pattern recognition receptors, cytokine networks, and intracellular kinase cascades