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The findings suggest that Volvalerenic A, the primary metabolite in KYXC, can enhance the accumulation of lipids, mitigate the inflammatory milieu, alleviate mitochondrial damage, and eventually impede the progression of atherosclerosis by activating the PGC-1/Sirt3/Epac1 signaling pathway

Effect persistence monitoring: because effects outlast serum presence, endpoint timing should reflect biological process kinetics (days-weeks) rather than serum PK kinetics (hours)

Morphology-independent virulence of Candida Species during Polymicrobial intra-abdominal infections with Staphylococcus aureus

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