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I'd add an evolutionary lens to this discussion - if we consider that humans are hypercarnivores based on our digestive anatomy and evolutionary history, "insulin resistance" becomes less about pathology and more about normal physiology responding to an unnatural food environment

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reported an increase in the levels of dysfunctional mitochondria and mitochondrial membrane proteins, translocase of the outer mitochondrial membrane 20 (Tom20), translocase of the inner mitochondrial membrane 23 (Tim23), and porin in the ASD temporal cortex without a change in the transcription of genes, such as PARK2 , responsible for the activation of mitophagy

doi: 10.3760/cma.j.cn112137-20221106-02333 102 van de VeerdonkFLBrggemannRJMVosSDe HertoghGWautersJReijersMHEet al