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Our data demonstrate that a common mitochondrial uncoupler dinitrophenol, which extends the mouse lifespan 36 , induces tbb-6 expression similarly as does acivicin, further indicating that both chemicals target ETC (Fig

Of these, arm1, in which residues S17, I18, V21 and A22 were deleted, while still containing G19 and P20 at the -hairpin tip, had no significant effect on the activity of Lgt, that is, colonies were formed normally in the complementation assay (Supplementary Figs 3c and 4a)

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