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Additionally, most ferroptosis modulators (e.g., ferrostatin-1 and triptolide) are evaluated in preclinical models of early AKI, but their efficacy in patients with established CKD (e.g., with tubulointerstitial fibrosis) is untestedwhether inhibiting ferroptosis can reverse or halt existing fibrosis, or only delay progression from AKI, remains unclear

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Melanotan 2 is a synthetic analogue of the human hormone -MSH (alpha-melanocyte stimulating hormone)

Thus, the interplay between oxidative stress, inflammation, and mitochondrial biogenesis represents a central axis in the pathophysiology of renal injury, underscoring the need for therapeutic strategies targeting these processes