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EA at ST36 and LI11 prevented neuroinflammation by regulating the miR-9-mediated NF-B signaling pathway in the ischemic cortex (116)

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These HMOs affect pathogen-associated molecular pattern signaling pathways [Toll-like receptor (TLR) 3, TLR5, IL-1, or cluster of differentiation 14 (CD14) expression and binding] decreasing cytokine production and the inflammatory response (56, 57)